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Absence of Enhancement of Spontaneous Echocardiographic Contrast by Thrombocytosis in a Patient with Left Ventricular Aneurysm, Primary Thrombocythemia, and von Recklinghausen Neurofibromatosis: A Case Report

Intensive Care Unit and the Emergency Room, Hôpital Saint-Louis, Paris, France

Michel Janier

Intensive Care Unit and the Emergency Room, Hôpital Saint-Louis, Paris, France

Alix Nguyen Van-Cao

Intensive Care Unit and the Emergency Room, Hôpital Saint-Louis, Paris, France

Jean-Roger Le Gall

Intensive Care Unit and the Emergency Room, Hôpital Saint-Louis, Paris, France

A thirty-eight-year-old man with primary thrombocythemia, von Reck linghausen neurofibromatosis, and myocardial-infarction-related left ventricu lar aneurysm with spontaneous echocardiographic contrast was followed up, suggesting that:

1. Neurofibromatosis may promote silent myocardial infarction or ischemia. Whether involvement of cardiac sensory nerves is a possible underlying mechanism remains nevertheless uncertain.

2. Platelets, whose role in the genesis of spontaneous echocardiographic con trast has been advocated, are probably not involved in this phenomenon, even in large numbers.

Angiology, Vol. 44, No. 8, 651-654 (1993)
DOI: 10.1177/000331979304400810


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