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Restrictive Cardiomyopathy Following Acute Myocarditis—A Case ReportDirector, Departments of Cardiology and Internal Medicine, Nagasaki Citizens Hospital, Nagasaki City, Japan
Department of Internal Medicine, Nagasaki Citizens Hospital, Nagasaki City, Japan
Department of Internal Medicine, Nagasaki Citizens Hospital, Nagasaki City, Japan
Department of Internal Medicine, Nagasaki Citizens Hospital, Nagasaki City, Japan
Department of Pathology, Nagasaki Citizens Hospital, Nagasaki City, Japan A fifty-five-year-old man developed intractable heart failure four weeks after upper respiratory infection. His central venous pressure was as high as 300 mmH2O. Two-dimensional echocardiogram revealed no muscular hypertrophy of the ventricles, no pericardial thickness, and no pericardial effusion. On catheterization, hemodynamic data were compatible with restrictive cardiomyopathy. Transvenous endomyocardial biopsy of the left ventricle was performed. Histologic examination strongly suggested that cardiomyopathy developed after acute myocarditis.
Angiology, Vol. 41, No. 1,
76-81 (1990) |
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